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Voltage-Gated Sodium Channel Blockers Library from OTAVA - OTAVA Ltd offers new Voltage-gated sodium channel blockers library containing 1630 compounds - OtavaChemicals.com
Voltage-Gated Sodium Channel Blockers Library from OTAVA

 

NewswireToday - /newswire/ - Toronto, Ontario, Canada, 2014/10/22 - OTAVA Ltd offers new Voltage-gated sodium channel blockers library containing 1630 compounds - OtavaChemicals.com.

   
 
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Voltage-gated sodium channels are a class of large integral membrane proteins, composed of a highly processed α subunit and one or several smaller β subunits. The α-subunit forms ion-conducting aqueous pore whereas auxiliary β subunits modify the kinetics and voltage-dependence of channel gating.

Voltage-gated ion channels are implicated in the regulation of synaptic transmission, muscle contraction and hormone secretion in response to membrane depolarization.
It was demonstrated that dysfunction of voltage-gated sodium channels led to the development of a wide range of human pathologies such as inherited epilepsy, migraine, periodic paralysis, cardiac arrhythmia, chronic pain syndromes and others. Therefore, the inhibition of voltage-gated sodium channels could be an effective strategy to prevent the development of these diseases.
OTAVA Ltd offers new Voltage-gated sodium channel blockers library containing 1630 compounds.

The library was designed as a special screening collection comprising compounds with predicted sodium channels blocking activity and selectivity. The compounds have been selected by pharmacophore screening of OTAVA Drug-like Green Collection toward three ligand-based pharmacophore models. The models were generated based on the known sodium channel blockers divided into three groups according to their structural features.

This library comprises drug-like compounds only and provides an excellent basis for drug discovery.

 
 
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Voltage-Gated Sodium Channel Blockers Library from OTAVA

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Publisher Contact: Andrey Dmytrenko - OtavaChemicals.com 
416-549-8030 info[.]otavachemicals.com
 
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